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While GHK-Cu treatment alone did not cause any visible histological changes or toxicity, pretreatment with GHK-Cu markedly attenuated these pathological alterations, resulting in a significantly reduced lung injury score compared to the LPS-only group

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This dual preservation of both NAD+ and methylation resources represents a unique mechanism among metabolic modulators, positioning 5-amino-1MQ as a bridge between energy metabolism and epigenetic regulation

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