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Myofibroblasts, the key effector cells in fibrosis, arise from fibroblasts, pericytes, endothelial cells, or epithelial cells via epithelial–mesenchymal transition (EMT) under stimulation by transforming growth factor (TGF) -β, clotting factors, and mechanical stress [11, 29, 34, 36]

Health Need - Immune Health

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These modifications significantly reduce its binding affinity for IGF-binding proteins (IGFBPs), extending its half-life from approximately 10 minutes (native IGF-1) to approximately 20–30 hours